What antigens can be found in body secretions if this phenotype is present: Oh, Le(a+b-)?
The ER orders 2 units of blood on patient, TJ. He has two antibodies, anti-E and anti-K. How many units will Blood Bank have to screen to be able to fill the order?
Patients with M pneumoniae infections are associated with what specificity of i?
I am a blood group system that is not intrinsic to RBCs, I attach at the C4 component of complement. My antigens are mostly destroyed ficin and papain but are resistant to treatment with DTT. Neutralization is often used to identify my antibodies.
Kernicterus is a build up of what?
My antibody specificity is usually anti-P and is only demonstrable in the Donath-Landsteiner test.
I am a blood group system that consists of 32 different antigens. I am only found on RBCs. I am well developed at birth so HDFN is a concern.
My serum has altered albumin to globulin ratio. However I usually do not interfere with the AHG phase of testing.
I can be found in some patients suffering a pneumonia infection with a titer level higher than 64.
What antigens will be present in body secretions if this phenotype is present: A,B,H, Le(a-b-)?
What ethnicity is most associated with Del?
What antigens can be found in body secretions if this phenotype is present: A,B,H, Le(a-b-)?
Verticales
My antigen group is carried on the Band 3 of the RBC membrane. HDFN is a concern.
I am a blood group system that is passively adsorbed onto RBC membranes from glycolipids present in plasma. (I am the undercover agent of blood group antigens).
I can cause problems in pregnancy b/c I decline dramatically, making the woman transiently have a neg phenotype, which can produce my antibodies. After delivery, her antigens return and the antibodies will disappear.
I am enhanced by ficin.
Neutralization is effective.
KEL 6 is almost completely confined to people of what ethnicity?
A Kleihauer-Betke test indicates 10 fetal cells per 1000 adult cells. For a women with 5,000 mL blood volume, how many vials of RhIg will she need?
Most weak D occurs in what ethnicity?
My system is hard to detect and are a common cause of delayed HTRs.
What is the most common partial D type in people of European descent?
If I am given in massive amounts, I can cause hemolysis through drug adsorption.
My antibodies are sensitive to enzyme treatment.
Does not bind
complement.
AHG and 37C.
IgG.
Shows dosage.
Glycoprotein on the red cell membrane that
serves as a RBC receptor for chemokines.
Expressed on cord blood cells.
If your patient was O+ and inherited FUT3 AND FUT2 genes, what would most likely be their ethnicity?
I am a blood group system associated with the resistance to P. vivax malaria?
I am a rare, autosomal recessive phenotype that lacks all ABH antigens.
What is the likely ethnic background of a person with a haplotype of ce?
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