The _____ trial is comparing cetuximab in combination with FOLFOX4 to FOLFOX4 alone.
Patients who developed acneiform rashes on cetuximab were more likely to _____ to treatment and more likely to survive longer.
Ctuximab is a human/murine chimeric _____ antibody.
Preclinical studies have demonstrated that cetuximab binds to the EGFR with high _____.
One possible mechanism is that development of the rash corresponds with _____ of EGFR.
Cetuximab has demonstrable activity in a variety of tumors and has been shown to circumvent _____ and enhance the effects of chemotherapy and radiotherapy.
The _____ trial is comparing cetuximab in combination with irinotecan to irinotecan alone.
The acneiform rash commonly develops within the first ____ weeks of treatment.
In a phase III trial, cetuximab in combination with cisplatin more than _____ the response rates.
Verticales
Activation of these pathways leads to _____ proliferation.
Nine patients whose tumors were EGFR negative and treatment with cetuximab led to a _____ in one patient.
EGFR is overexpressed in a significant proportion of glioblastomas, breast, _____, colorectal, head and neck, bladder, ovarian and prostate cancers.
The epidermal growth factor receptor(EGFR) is commonly _______ in a variety of solid tumors.
Cetuximab is an _____ treatment in tumors expressing EGFR.
In a phase I/II study, cetuximab was administered with _____ and carboplatin.
Overactivation of EGFR signalling may be driven by an _____ secretory loop involving overproduction of ligands.
From these studies, the main side effects of cetuximab monotherapy were an _____ rash, allergic reactions and lethargy.
EGFR was the first _____ identified as part of a family of receptor tyrosine kinases.
The observed response rate with cetuximab in combination with irinotecan is _____ to that seen with oxaliplatin combined with 5FU in the second line treatment.
The input layer consists of _____ of which EGF and transforming growth factor alpha are recognized as the most important ligands for EGFR.
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